Primary Headaches - Cluster Headache Prevention - The Do's and Do not's list

I. Cluster Headache
Cluster Headache also known as suicide headache, is defined as an uncommon distinctive neurovascular syndrome occurring in either episodic or chronic patterns of that occur periodically over a long period of time. The diseases affects over 0.1% of the population, occurring at 45-to 60-day intervals with one to three headaches a day lasting 45 min to 2 h(a), if untreated it, can cause increased frequency of the attacks.

II. Prevention
A. Do's and Do not's list, if you are experience migraine headache because of foods, food additives, chemical compounds, alcohol, smoking, stress, life style, etc.
1. Food elimination diet

Certain foods which trigger the over production of IgG antibodies if eliminated from the diet can result in the decreased number of migraine like headaches over 4weeks, some studies suggested that using the ELISA test with subsequent diet elimination advice significantly reduce the number of migraine like headaches at 4 weeks.(34)

2. Diet restriction
Diet restriction based on IgG antibodies is an effective strategy in reducing the frequency of migraine attacks. In the study conducted by Istanbul Faculty of Medicine, Department of Neurology, Istanbul, Turkey in a 6-week baseline, IgG antibodies against 266 food antigens were detected by ELISA(35)

3. Foods and chemical compounds trigger migraine headache
a. Certain foods can trigger the migraine headache attack through an allergic reaction. Citrus fruits, tea, coffee, pork, chocolate, milk, nuts, vegetables and cola drinks have been cited as possible allergens associated with migraine. If you are experience the attack after taking some of those, the best way is to avoid them. Researchers also suggested that substances are tyramine, phenylalanine, phenolic flavonoids, alcohol, food additives (sodium nitrate, monosodium glutamate, aspartame) and caffeine may be the cause of modifications in vascular tone and bring migraine on(36)

4. Alcohol
As we all known that a small dose of alcohol increase the protective effect on cardiovascular disease, but excessive drinking can increase the frequency of migraine headache as alcoholic drinks are a migraine trigger in about one third of patients with migraine in retrospective studies on trigger factors(37)

5. Smoking
There is evidence that migraine was associated with several lifestyle and socioeconomic factors, In the investigation conducted by University of Copenhagen with a questionnaire containing validated questions to diagnose migraine and questions on lifestyle and socioeconomic factors was sent to 46,418 twin individuals residing in Denmark. 31,865 twin individuals aged 20-71 and most associations such as low education and employment status were probably due to the negative effects of having migraine while others such as smoking were risk factors for migraine.(38)

6. Others may trigger headache migraine attacks to certain people, such as
a. Too much work (Stress)
b. Too little sleep (Sleep related)
c. Passive smoking (Smoking)
d. Perfume (Chemical compound)
e. Etc.(39)

7. Moderate exercise
In a study of Forty women with general migraine attending the Neurology Department of the Faculty of Medicine Faculty of Dokuz Eylül University, regular long-term aerobic exercise is found in reduced migraine pain severity, frequency and duration possibly due to increased nitric oxide production.(40)

8. Management of migraine
Hygiene and behaviour measures capable of ensuring the best possible well-being (regular meals and balanced diet, restriction of alcohol and smoking, regular sleeping pattern, moderate physical exercise and relaxation) have found to reduce the frequent migraine attack(41)

9. Avoid medication overuse headache (MOH)

There are report that overuse of migraine medication can result of increased frequency to daily or near-daily as a rebound effect comes into play(42)

10. Etc.

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For the series of headaches/migraines, please visit http://diseases-researches.blogspot.ca/2012/01/series-of-article-of-headache.html


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Sources

(a) http://www.ncbi.nlm.nih.gov/pubmed/18047865  

(34) http://www.ncbi.nlm.nih.gov/pubmed/21835022
(35) http://www.ncbi.nlm.nih.gov/pubmed/20647174
(36) http://www.ncbi.nlm.nih.gov/pubmed/8681169
(37) http://www.ncbi.nlm.nih.gov/pubmed/21336550
(38) http://www.ncbi.nlm.nih.gov/pubmed/21390550
(39) http://www.ncbi.nlm.nih.gov/pubmed/20847084
(40) http://www.ncbi.nlm.nih.gov/pubmed/12971707
(41) http://www.ncbi.nlm.nih.gov/pubmed/20464586
(42) http://www.ncbi.nlm.nih.gov/pubmed/20464586